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Short-chain fatty acids (SCFA) are involved in immune system and inflammatory responses. We comprehensively assessed the host genetic and gut microbial contribution to a panel of eight serum and stool SCFAs in two cohorts (TwinsUK, n = 2507; ZOE PREDICT-1, n = 328), examined their postprandial changes and explored their links with chronic and acute inflammatory responses in healthy individuals and
This case note discusses Swedish Labour Court case No. 33 of 2023 (AD 2023:33). The case involved a fixed-term worker employed as a personal assistant under a specific form of fixed-term contract regulated by collective agreement. The trade union sought to have the employment contract converted into a contract of indefinite duration and the form of fixed-term employment used to be declared invalid
Diabetes is a highly heterogeneous condition; yet, it is diagnosed by measuring a single blood-borne metabolite, glucose, irrespective of aetiology. Although pragmatically helpful, disease classification can become complex and limit advances in research and medical care. Here, we describe diabetes heterogeneity, highlighting recent approaches that could facilitate management by integrating three d
Studies on fibre intake and incident colorectal cancer (CRC) indicate inverse associations. Differences by tumour stage have not been examined. We examined associations between fibre intake and its sources, and incidental CRC. Separate analyses were carried out on the basis of sex, tumour location and the Tumour, Node, Metastasis (TNM) classification. The Malmö Diet and Cancer Study is a populatio
To study the prognosis of patients with newly diagnosed Type 2 diabetes in primary care in relation to their baseline C-peptide concentration.
The S100 alarmins A8, A9, and A8/A9, secreted by activated neutrophils and monocytes/macrophages, are involved in the pathogenesis of various inflammatory diseases. S100A8/A9 has previously been linked to atherogenesis and cardiovascular (CV) disease. We investigated whether S100A8, A9, and A8/A9 correlate with carotid artery disease and CV risk in apparently healthy individuals.
The majority of reported complex disease associations for common genetic variants have been identified through meta-analysis, a powerful approach that enables the use of large sample sizes while protecting against common artifacts due to population structure and repeated small-sample analyses sharing individual-level data. As the focus of genetic association studies shifts to rare variants, genes
The unclear link between intake of polyunsaturated fatty acids (PUFAs) and risk of cardiovascular disease (CVD) could depend on genetic differences between individuals. Minor alleles of single-nucleotide polymorphisms (SNPs) in the ∆5 fatty acid desaturase (FADS) 1 gene were associated with lower blood concentrations of long-chain ω-3 (n-3) and ω-6 (n-6) PUFAs, indicating an associated loss of fun
Lack or dysfunction of insulin producing β cells results in the development of type 1 and type 2 diabetes mellitus, respectively. Insulin secretion is controlled by metabolic stimuli (glucose, fatty acids), but also by monoamine neurotransmitters, like dopamine, serotonin, and norepinephrine. Intracellular monoamine levels are controlled by monoamine oxidases (Mao) A and B. Here we show that MaoA
Background: High plasma copeptin (copeptin), the C-terminal fragment of arginine vasopressin pro-hormone, has been associated with the metabolic syndrome (MetS), diabetes mellitus (DM) development and nephropathy. Here we tested whether elevated copeptin level is associated with later development of the MetS, its individual components and microalbuminuria. Methods: We analysed copeptin at baselin
We have previously shown that genetic variance in NEDD4L, a regulating protein of a sodium channel in the distal nephron, has been associated with marginally higher blood pressure and enhanced salt sensitivity. Here, we tested if the genetic NEDD4L variation previously associated with salt sensitivity is related to population blood pressure, incidence of cardiovascular disease (CVD) and mortality.
Ny rättspraxis från EG-domstolen i fallen Köbler och Kühne & Heitz har visat att domstolen är beredd att etablera rättsprinciper som kommer göra det möjligt att på ett effektivt sätt hantera missbruket av acte clair-doktrinen. I det förstnämnda fallet, etablerade domstolen en möjlighet att utkräva skadeståndsansvar av medlemsstaten i det fall den nationella sista instansrätten (in casu den hög
Rationale: Atherosclerotic plaques that give rise to acute clinical symptoms are typically characterized by degradation of the connective tissue and plaque rupture. Experimental studies have shown that mechanisms to repair vulnerable lesions exist, but the rate of remodeling of human plaque tissue has not been studied. Objective: In the present study, we determined the biological age of different
Next-generation sequencing technologies are making it possible to study the role of rare variants in human disease. Many studies balance statistical power with cost-effectiveness by (a) sampling from phenotypic extremes and (b) utilizing a two-stage design. Two-stage designs include a broad-based discovery phase and selection of a subset of potential causal genes/variants to be further examined in
Background Plasma triglyceride levels are heritable and are correlated with the risk of coronary heart disease. Sequencing of the protein-coding regions of the human genome (the exome) has the potential to identify rare mutations that have a large effect on phenotype. Methods We sequenced the protein-coding regions of 18,666 genes in each of 3734 participants of European or African ancestry in the
Lund Psychological Reports Volume 12, No. 2, 2012 A measure of internal and external motivation to control in-group bias Øyvind Jørgensen, Martin Bäckström and Fredrik Björklund Department of Psychology, Lund University, Sweden Lund Psychological Reports Editor: Magnus Lindgren ISSN 1404-8035 2 Abstract A measure of individual differences in the motivation to control in-group bias (favouritism) wa
https://www.psy.lu.se/sites/psy.lu.se/files/prvol12no2.pdf - 2026-07-21
U ni ve rs ite ts bi bl io te ke ts u ts tä lln in gs ka ta lo g 64 Universitetsbiblioteket, Lunds Universitet Box 3, 221 00 Lund UB Media ISSN 0281-3823 LULID/LIBR - 1547 kris, Kitsch OCH KASSLER MED ANANAS Utställning på Universitetsbiblioteket i Lund 4 maj - 27 september 2012 U tställningsproduktion: H åkan H åkansson, K ristian K nutsson, M ikael Lindgren, B engt M elliander, A nders N avalliu
https://www.ub.lu.se/en/sites/ub.lu.se.en/files/2012_80-tal.pdf - 2026-07-21
Project code: 1 SGFF – abridged Style sheet December 2014 Author(s) AU AN AUTHOR and ANOTHER AUTHOR Authors are listed by full name only. Ranged left. e.g. JANICE BLACK JANICE BLACK and FRANCES FABIAN JANICE BLACK, FRANCES FABIAN, KIM HINRICHS and FIONA LEE EMILY DARLING, KRISTINE MOLINA, MELISSA SANDERS, FIONA LEE and YU ZHAO Affiliation AFF Multiple email addresses where multiple authors share t
https://www.geologi.lu.se/sites/geologi.lu.se/files/geol01_-_gff_style_sheet_authors_dec_2014.pdf - 2026-07-21
1 Utvärdering av forskarutbildningen i socialt arbete vid Lunds universitet 2019 Undertecknande sakkunniggrupp vill härmed avlägga följande utlåtande gällande utvärdering av forskarutbildningen i socialt arbete vid Lunds universitet. Sakkunniggruppen har bestått av: Peter Dellgran, professor i socialt arbete vid Göteborgs universitet (ordförande) Marjaana Seppänen, professor i socialt arbete vid H
https://www.sam.lu.se/internt/sites/sam.lu.se.internt/files/sakkunnigutlatande_utvfoutbsocarbfinal.pdf - 2026-07-21
Avaya one-X™ Deskphone Edition for 9630 IP Telephone User Guide Avaya one-X™ Deskphone Edition for 9630 IP Telephone User Guide 16-300700 Issue 3 January 2007 Contents Notices ..................................................................................................... 5 Introduction to the 9630/9630G IP Telephone ...................................... 7 Overview ..........................
https://www.it.lu.se/sites/it.lu.se/files/telefoni_avaya_9630_manual_eng.pdf - 2026-07-21
